Talk:Mad cow

Concerning the content as posted, requires some critical analysis pertaining to the uses of transgenic plants in conjunction with xenobiotics. And animals insects with human control dietary sources, educational ontological thesis resources, and the observed inability to discriminate the likely alternate educational structure anti-thesis evolved to make sustainable populations and resources available for the notion that they represent recruiting/education of the at risk population when compared to establishing the UDP drug metabolism and dietary intake in compromised individual populations necessary, or just as well becoming the growing belief in biological determinism and awareness could become disproportionate to the plethoric feedback that may vary. 7/23/2007

Transgenic animals and plants in diets of humans, with xenopbiotic, glucotoxicity further deposition of (in) fat in the liver and muscle, causing local tissue insulin resistance, as well as a rise in circulating factors that are atherogenic [ low-methelated germ-lines produced or possible to produce/ Or a improbable coding non-methylated copy aneuploidism~[uniparental disomy (UPD)]~(disease) have poorly understood control populations dietary intake/compared to establishing the drug metabolism, [since “hazardous aggregates of proteins survive digestion and are distributed throughout the human body.”[[[1]{this a case of unstable Bt-Transgenic animals and plants }July 17-2007]] where the zona plucida has lost in the subjective ecological threshold (student-T-test [recruiting the at risk population]) competition barrier of typical phase I-II drug-metabolizing enzymes, or loses the ability to discriminate the blood brain barrier partition and control the in the cell cortex of the major lymph system roles where the UGT1A1 genotype [UDP glucuronosyltransferase 1 family] is particularly for a drug metabolism/ to the [2] “TSS” transcription start site; Statistically, and so no logical name or collections of gene locus autologus to ie. Chromosome 5‘-3‘,p-q candidate gene pair Plausibility hypotheses, to the etiological (20th century concepts), distributions [complexity] which are highly conserved in mammalian and insect on the human chromosomes 19p-q13.3. Is located directly adjacent to a gene that is unrelated at region on 19q13.3 in human though (pure and complicated autosomal recessive) eg. associated with non-statistically significant RR relative risk reductions, at this time in female X chromosme dominant inheritance (mostly excluded) are not included in out-breeding mammalian genetic studies. To the best-fitting model codominant mendelian inheritance predominant redox-signal, the calculated redox state control is subordinate to signals which define the assimilatory force, in a comparison of normotensive subjects in an examination of the biochemical processes occur at an early stage in ontogeny; and horizontal gene mutational in adult development, related to productive activity’s dominant theme (histocompatiblity in non-transgenic plants-animals is when not a rare disorder is associated with the centromere instability. ) and recognition of genomic features. [uni-vackvsug] FAROUCHE OMBRÉ LLC [3] July 23, 2007

Media:http://www.prwatch.org/node/6108#comment